3 edition of Macrophage-pathogen interactions found in the catalog.
Includes bibliographical references and index.
|Statement||edited by Bruce S. Zwilling, Toby K. Eisenstein.|
|Series||Immunology series ;, 60, Immunology series ;, v. 60.|
|Contributions||Zwilling, Bruce S., 1943-, Eisenstein, Toby K.|
|LC Classifications||QR185.8.M3 M2947 1994|
|The Physical Object|
|Pagination||xx, 634 p. :|
|Number of Pages||634|
|LC Control Number||93027941|
The gene encoding the natural resistance-associated macrophage protein 1 (Nramp1) is one of the most extensively characterized mouse host resistance loci. Initially it was observed that certain inbred mouse strains were innately susceptible to infection with the intracellular pathogens Salmonella typhimurium, Leishmania donovani and Mycobacterium bovis (bacille Calmette-Guérin; reviewed in ). NutrImmune 26Y are self-affirmed GRAS supplements that provide IgY immunoglobulins and immunoregulating molecules from hyperimmune chicken egg. Research suggests that this dairy-free source of immunoglobulins and immune cofactors helps support immune function in the intestine and a healthy intestinal environment. He worked as a post-doctoral scientist in the laboratory of Prof Malcolm McConville (University of Melbourne) before joining the NHMRC Program on Cellular Microbiology headed by Prof Trevor Lithgow, Monash University. In he established the macrophage-pathogen interaction group with funding from the NHMRC. Xenografts of the hematopoietic system are extremely useful as disease models and for translational research. Zebrafish xenografts have been widely used to monitor blood cancer cell dissemination and homing due to the optical clarity of embryos and larvae, which allow unrestricted in vivo visualization of migratory events. Here, we have developed a xenotransplantation technique that.
Mycobacterium tuberculosis (Mtb) is an intracellular pathogen that infects alveolar macrophages following aerosol transmission. Lung macrophages provide a critical intracellular niche that is required for Mtb to establish infection in the human host. This parasitic relationship is made possible by the capacity of Mtb to block phagosome maturation following entry into the host macrophage.
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Print book: EnglishView all editions and formats Summary: Examines the complex relationships between mononuclear phagocytes and the infectious agents that live and replicate within them, describing the basic characteristics of macrophages, including their differentiation, activation, antigen processing and presentation, and antimicrobial mechanisms.
This excellent book introduces basic and recent Macrophage-pathogen interactions book concepts regarding macrophage-biology and macrophage-pathogen interactions in a attractive and clear manner of presentation. Therefore it is recommended for postgraduates and researchers in the area of Immunology and Infectious Diseases.
Myrthes Toledo Barros. Genre/Form: Electronic books: Additional Physical Format: Print version: Macrophage-pathogen interactions. New York: M. Dekker, © (DLC) Macrophage-pathogen interactions in infectious diseases: new therapeutic insights from the zebrafish host model Vincenzo Torraca, Samrah Masud, Herman P.
Spaink, and Annemarie H. Meijer * Institute of Biology, Leiden University, Einstein CC, Leiden, The Netherlands.
Macrophage-pathogen interactions in infectious diseases: new therapeutic insights from the zebrafish host model. Torraca V(1), Masud S(1), Spaink HP(1), Meijer AH(2).
Author information: (1)Institute of Biology, Leiden University, Einstein CC, Leiden, The Netherlands. This chapter discusses specific issues in macrophage–pathogen interactions that compare different strategies adapted by individual pathogens to achieve a similar endpoint within the host. This chapter focuses on the pathogen strategies that is a successful intracellular pathogen must be able to enter a host cell, survive, and preferably replicate therein.
In the context of macrophage–intracellular bacteria interactions, the plasma membrane of immune cells will be the first barrier the invading pathogen encounters.
To successfully infect the host, pathogens often evolve various strategies to target the lipid-enriched plasma membrane for entry and exit, or to hijack host lipid metabolism to. Buy Macrophage-Pathogen Interactions (Immunology) 1 by Zwilling, Bruce S., Eisenstein, Toby K.
(ISBN: ) from Amazon's Book Store. Everyday low prices and free delivery on Format: Hardcover. Research: Aim: The aim of the laboratory is to discover the molecular and cellular interactions that allow human pathogens to survive in macrophages.
Perspective: Macrophages are phagocytic (to engulf and digest) immune cells designed to kill invading microorganisms by employing a diverse arsenal of anti-microbial agents. They are part of the first line defence (innate immune response) but. Krahenbuhl, J. L., and Adams, L.
B.,The role of the macrophage in resistance to the leprosy bacillus, in: Macrophage-Pathogen Interactions (B. Zwilling and T. Eisenstein, eds.), Marcel Dekker, New York, pp. – Over the past decade of M.
tuberculosis research, the means to manipulate both the organism and the host has ushered in an exciting time that has uncovered some of the mechanisms of the innate macrophage-pathogen interactions that lie at the heart of Macrophage-pathogen interactions book.
tuberculosis pathogenesis, though many interactions likely still await discovery. Macrophage-pathogen interactions. New York: Mar- cel Dekker Inc. Integrates macrophage immunobiology and interactions with a range of bacte- ria, parasites, fungi and Macrophage-pathogen interactions book.
Individual chapters Macrophage-pathogen interactions book a wide range of review articles, as well as key papers of modem observations. Macrophage-Pathogen Interactions edited by Bruce S. Zwilling and Toby K.
Eisenstein Dekker (Immunology Series), $ hbk (xx + pages) ISBN 0 X This book is part of the Immunology series edited by Noel R. Rose. Part of the Infectious Agents and Pathogenesis book series (IAPA) Keywords C. L., and Winter, A. J.,Macrophages and Brucella, in: Macrophage-Pathogen Interactions (B.
S., Zwilling and T. KThe immune response to Brucella abortus — the cell-mediated response to infections, in: Animal Brucellosis (K. Nielsen and J. Cells, an international, peer-reviewed Open Access journal.
Cryptococcus has a polysaccharide capsule, which is the major virulence factor of the organism. This chapter covers the interactions of Cryptococcus with immune cells, with a focus on macrophages. Macrophages produce cytokines and chemokines, thereby recruiting and activating other immune cells such as T cells and neutrophils.
In vitro studies have shed important light on macrophage. This chapter discusses specific issues in macrophage–pathogen interactions that compare different strategies adapted by individual pathogens to achieve a similar endpoint within the host.
Abramson, S. L, and Gallin, J. I.,IL-4 inhibits superoxide production by human mononuclear phagocytes. Immunol. – PubMed Google Scholar. Understanding macrophage–pathogen interactions is crucial to understanding the pathogenesis of many infectious diseases.
The balance between the macrophage’s ability to recognize bacterial pathogens and the pathogen’s ability to modulate macrophage signalling often determines the outcome of an is shown. Koch, B.E., Stougaard, J., Spaink, H.P.
() Keeping track of the growing number of biological functions of chitin and its interaction partners in biomedical research. Glycobiology. 25(5) RAW cells may be considered as a cell model for study on interactions between E. tarda and macrophages. MOIs of and are recommended for using E. tarda-invaded RAW cells.
tarda could induce the apoptosis in RAW cells through the involvement of caspase Once inside macrophage, numberous E.
tarda could be destroyed within the phagolysosomes. Find many great new & used options and get the best deals for Immunology: Macrophage-Pathogen Interactions Vol. 60 (, Hardcover) at the best online prices at eBay.
Free shipping for many products. Pneumonia caused by Streptococcus pneumoniae is a consequence of failure by the host to clear or kill pneumococci inhaled into the lung. The alveolar macrophage is the resident phagocyte of the alveoli of normal human lungs and is responsible for the removal of pathogens that enter gas-exchanging areas.
In book: Model Organisms for Microbial Pathogenesis, Biofilm Formation and Antimicrobial Drug Discovery, pp Macrophage-pathogen interactions in infectious diseases: New therapeutic.
Mouse peritoneal and alveolar macrophages were interacted in vitro with C. parvum oocysts and cultured in normal medium and in medium with IFN-γ. The results showed that in vitro activation of macrophages by IFN-γ limits C.
parvum development although the inhibitory effect is not as potent as in other intracellular parasitic protozoa.
The interactions between components and limited views of the pathways they form have been described in detail in the literature used to construct this diagram (see interaction table, additional file 2).
An overview of the pathway biology depicted by the diagram. Manor, E.,The effect of monocyte-derived macrophages on the growth of Rickettsia conorii in permissive cells, in: Rickettsiae and Rickettsial Diseases, Proceedings of the Fourth International Symposium (J.
Kazar and D. Raoult, eds.), Publishing House of the Slovak Academy of Sciences, Bratislava, Czechoslovakia, pp. – Google Scholar. For the ecological classification, see Macrophage (ecology). Macrophages (Greek: big eaters, from Greek μακρός (makrós) = large, φαγεῖν (phagein) = to eat) are a type of white blood cell of the immune system, that engulfs and digests cellular debris, foreign substances, microbes, cancer cells, and anything else that does not.
Hu, W., Yang, S., Shimada, Y., Münch, M., Marín-Juez, R., Meijer, A.H., Spaink, H.P. () Infection and RNA-seq analysis of a zebrafish tlr2 mutant shows a broad. Abstract. Iron is essential for both host and pathogen, and complex systems of acquisition and utilization have evolved in competition.
Our increasing knowledge of the basic mechanisms of homeostasis and their adaptation during deficiency, overload, and infection indicate that iron is a key regulator of host pathogen interactions.
Macrophage-pathogen interactions in infectious diseases: new therapeutic insights from the zebrafish host model.
Dis Model Mech. ; – doi: /dmm [ PMC. The host-pathogen interaction is defined as how microbes or viruses sustain themselves within host organisms on a molecular, cellular, organismal or population level. This term is most commonly used to refer to disease-causing microorganisms although they may not cause illness in all hosts.
Because of this, the definition has been expanded to how known pathogens survive within their host. This chapter describes the major mechanisms of killing used by the macrophage, how innate killing mechanisms, immune-regulated killing mechanisms can be modulated by the host immune response, and how different bacteria have learned to deal with this potential nemesis.
Macrophages are extremely degradative cells. One of their prime tasks while they migrate through body tissues is to recognize. Citation: Go N, Bidot C, Belloc C, Touzeau S () Integrative Model of the Immune Response to a Pulmonary Macrophage Infection: What Determines the Infection Duration.
PLoS ONE 9(9): e Overall emphasis is on understanding the mechanisms of host-pathogen interactions rather than on specific research approaches. Brogden is affiliated with the US Department of Agriculture. Material originated at a September conference.
Annotation c. Book News, Inc., Portland, OR () BooknewsPrice: $ Introduction. A hallmark of infections with certain viruses (e.g., herpesviruses), intracellular bacteria (e.g., Mycobacteria, Coxiella, Chlamydiae), or protozoa (e.g., Trypanosoma cruzi, Leishmania) is the long-term persistence of the pathogen after clinical cure of the on in vitro results, modulation of host cell antimicrobial activities, synthesis of inhibitory cytokines.
Vitamin D and immune system Article Literature Review (PDF Available) in Journal of Endocrinology (2) March with 53 Reads How we measure 'reads'.
Meaning and Truth in Habermas's Pragmatics. Here we review the history and current state of the art of this topic and we offer a model of the macrophage-pathogen interaction that takes into. Here we review the history and current state of the art of this topic and we offer a model of the macrophage-pathogen interaction that takes into the account the complexities of programmed cell death and the relationship between various death signaling pathways and host defense in TB.
"Macrophage-Pathogen Interactions," B.S. Zwilling and T.K. Eisenstein (eds.). Marcel Dekker, Inc., New York. "The Brain Immune Axis and Substance Abuse," B. Sharp, T.K. Eisenstein, J. Madden, and H. Friedman (eds.).
Plenum Press, New York. "AIDS, Drugs of. Inflammasomes are cytosolic multiprotein complexes that regulate inflammatory responses to danger stimuli and infection, and their dysregulation is associated with an increasing number of autoinflammatory diseases.
In recent years, zebrafish models of human pathologies to study inflammasome function in vivo have started to emerge. Here, we discuss inflammasome research in zebrafish in light of.Macrophage-pathogen interactions in infectious diseases: new therapeutic insights from the zebrafish host model.
Dis Model Mech ; 7: Dis Model Mech ; 7: Xiao D, Xiu Y, Yue H, Sun X, Zhao H, Liu S.Research Publications. Medicine Books and Chapters. Beaman, M.H., Subauste, C.S., Wong, S.Y. and Remington, J.S. Macrophage-Toxoplasma interactions, Macrophage.